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Kaitlyn Bader is the assistant managing editor of 

 and joined the MJH Life Sciences team in June 2022. She attended Otterbein University in Westerville, Ohio, and earned a Bachelor of Arts degree in literary studies with minors in creative writing and film studies. Prior to health care news, Kaitlyn worked in industrial marketing. She enjoys cooking, reading, and tending to her house plants.

Articles

In case you missed it, this week we had news about phase 2 initiation of EVO756 for chronic inducible urticaria, IBI363's Fast Track Designation for advanced melanoma, Arcutis' enrollment of the last patient in ARQ-255 trial for alopecia areata, and more.

The Weekly Roundup: September 2-6

Maddi Hebebrand is an associate editor of 

and joined the MJH Life Sciences team in May 2024. She attended Baldwin Wallace University, studying Media Production and Film, and is now finishing her Masters in Digital Media through Ohio University. When she's not writing, Maddi loves to read, attend concerts and spend time with her family.

A recent study investigated the acceptability, barriers, and facilitators associated with digital health tools.

Exploring the Perception of Digital Health Tools in Dermatology

The JAK1 inhibitor provided significant relief for 2 patients with refractory PN.

Abrocitinib Shows Promise for Prurigo Nodularis

and joined the MJH Life Sciences team in February 2023. She attended Kent State University and earned a Bachelor of Science degree in journalism with minors in public relations and creative writing. She enjoys making and listening to music, reading, and spending time with her cats, Astrid and Marceline.

Researchers conducted a web-based survey to gain insights into current clinical practice for PN in Japan. 

Approximately 65% of Dermatologists in Japan Report Dissatisfaction With Treatment Options for Severe Prurigo Nodularis

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Boston — New research discoveries demonstrate that melanoma is highly dependent on cyclin-dependent kinase 2 (CDK2) for growth and proliferation. Inhibiting expression of the gene regulating CDK2 stops the cancer dead in its tracks.

New target, intervention for treating melanoma